Lysol is a disinfectant designed for use on surfaces and contains phenol, a protoplasmic poison. Poisoning from Lysol can occur through oral ingestion, skin contact, inhalation, and other means. Phenol is mainly metabolized by the kidneys, and after absorption, it causes widespread capillary damage and clotting in superficial blood vessels. This results in urine that is scanty with traces of albumin, and microscopically, blood and calcium oxalate crystals are observed. The urine turns olive green or brown on standing, a phenomenon called carboluria.Studies by J. Jyrkkanen 2020 on antibiotic impacts on mitochondria showed that ROS damage and ruptures of mitochondria resulted in pathology leading to loss of oxidative phosphorylation, likelihood of increase in cancers favorite metabolism aerobic glycolysis, suppression of the p53 DNA repair gene, cardiovascular ailments and immune dysfunction. The similarity of damage reported here by google AI suggest a similar pathology for Lysol type antibiotic cleaners.Otherwise damaged mitochondria induced effects have been observed also for some common pesticides, and mRNA vaccine.The problem arises from the fact that Lysol is an antibiotic that kills bacteria and mitochondria are a highly modified essential symbiotic derivative of an ancient bacteria.
Characteristics
Values
Sensation
Constriction around the throat, burning pain from the mouth to the epigastrium
Taste
Sour and acidic
Urine
Scanty, with traces of albumin
Microscopic Observations
Blood and calcium oxalate crystals
Other Observations
Burns on the face and skin, mucosa of the mouth, tongue, pharynx, and esophagus may be bleached
Phenol Metabolism
Converted into hydroquinone and pyrocatechol, then excreted in urine
Urine Color
Olive green or brown on standing (carboluria)
Impacts on Mitochondria
The active ingredient benzalkonium chloride reveal a pattern of cellular toxicity mediated by mitochondrial damage.
Disruption of Energy Production
QACs which are commonly used in antimicrobial products, have been shown to inhibit mitochondrial respiration specifically at Complex I of the electron transport chain, leading to a decrease in cellular energy (ATP)
Structural Damage or Fragmentation
Exposure to these compounds causes structural disruption, resulting in mitochondrial fragmentation and changes in mitochondrial membrane potential
Induction of Oxidative Stress
Similar to other chemical sanitizers, these compounds can induce the generation of reactive oxygen species (ROS) within the mitochondria, causing further impairment.
Initiation of Apoptosis
Mitochondrial dysfunction caused by these surfactants can trigger the mitochondrial pathway of apoptosis or programmed cell death
Compounds with Documented Mitochondrial Toxicity
Quaternary Ammonium Compounds (QAC’s/BAC’s) induce mitochondrial dysfunction in epithelial cells
Cetylpyridinium Chloride (CPC)CPC causes severe nanostructural disruption, reduces ATP production, and acts as a mitochondrial inhibitor.
Ammonium Hydroxide a caustic compound present in some cleaners can cause severe irritation and potential cellular disruption.
CONCLUSION
These disinfectants can cause significant mitochondrial dysfunction in mammalian cells leading to significant morbidity and mortality. While disinfectant effect may reduce exposure to pathogenic microbes in the short term the harm to beneficial endogenous mitochondria may cause health issues in the long term.
References
Jyrkkanen jccm-aid1104
Jyrkkanen J. Antibiotic induced changes to mitochondria result in potential contributions to carcinogenesis, heart pathologies, other medical conditions and ecosystem risks. J Cardiol Cardiovasc Med. 2020; 5: 163-171
LINKS BETWEEN RF AND EMF AND BRAIN CANCERS. JORMA JYRKKANEN RESEARCHER 2025-03-25
Key Points
Research suggests a possible link between long-term exposure to electromagnetic fields (EMF) and radiofrequency (RF) radiation from cell towers and increased risk of brain cancers and Schwannomas, but evidence is not conclusive.
The International Agency for Research on Cancer (IARC) classifies RF radiation as “possibly carcinogenic to humans,” highlighting ongoing controversy.
Studies show mixed results, with some finding small increases in certain tumors, especially in animal studies, while human studies often show no clear association.
Avoidance guidelines include reducing call time, using speaker mode or headphones, and maintaining distance from cell towers, though effectiveness varies.
Overview
The connection between EMF, RF, and cell tower frequencies and brain cancers like gliomas and Schwannomas (tumors of nerve cells, including acoustic neuromas) is a complex and debated topic. While some studies suggest potential risks, especially with long-term exposure, the evidence is not definitive, and expert opinions differ.
Current Research Findings
Research leans toward a possible association, particularly with heavy, long-term use of cell phones and proximity to cell towers. For instance, the National Cancer Institute (NCI) notes stable brain tumor rates despite increased cell phone use, but animal studies, like those by the National Toxicology Program (NTP), found small increases in heart Schwannomas in male rats exposed to RF radiation (Cell Phones and Cancer Risk Fact Sheet – NCI). The IARC’s 2011 classification of RF as “possibly carcinogenic” reflects limited evidence, fueling ongoing debate.
Types of Cancers and Tumors
Brain cancers, such as gliomas, and Schwannomas, particularly acoustic neuromas, are of concern. Some studies, like the Interphone study, found a small increase in glioma risk for heavy cell phone users, while others, like the Danish Cohort, found no association. Schwannomas, linked to nerve cells, showed increased risk in animal studies but inconsistent results in humans.
Avoidance Guidelines
To minimize exposure, experts suggest reducing call duration, using speaker mode or wired headphones, avoiding calls in areas with weak signals (which increase emission), and maintaining distance from cell towers. The Food and Drug Administration (FDA) recommends these measures, though their impact on reducing cancer risk is not fully proven (Reducing Radio Frequency Exposure from Cell Phones – FDA).
Survey Note: Detailed Analysis of EMF, RF, and Cell Tower Frequencies Linked to Brain Cancers and Schwannomas, Including Avoidance Guidelines
This note provides a comprehensive overview of the relationship between electromagnetic fields (EMF), radiofrequency (RF) radiation, and cell tower frequencies with brain cancers and Schwannomas, including detailed findings from recent studies and practical avoidance guidelines. The analysis is grounded in current scientific literature, with a focus on clarity for a lay audience while maintaining technical accuracy.
Introduction
EMF and RF radiation are forms of non-ionizing electromagnetic radiation emitted by cell towers, cell phones, and other wireless devices. Cell towers operate at frequencies typically ranging from 700 MHz to 2.7 GHz for 2G, 3G, and 4G, and up to 80 GHz for 5G, as noted by the NCI (Cell Phones and Cancer Risk Fact Sheet – NCI). Concerns arise about potential links to brain cancers (e.g., gliomas, meningiomas) and Schwannomas, which are tumors of Schwann cells, often manifesting as acoustic neuromas. This note synthesizes findings from epidemiological, animal, and expert reviews, acknowledging the complexity and controversy surrounding the topic.
Scientific Evidence and Findings
Epidemiological Studies in Humans
Human studies provide mixed results, with no definitive link established. The NCI’s fact sheet, updated in April 2024, indicates stable incidence rates for adult gliomas, pediatric brain tumors, and acoustic neuromas in the US and other regions, despite increased cell phone use (Cell Phones and Cancer Risk Fact Sheet – NCI). Key studies include:
Danish Cohort Study: No association with glioma, meningioma, or acoustic neuroma, even for subscribers over 13 years, with ORs not significantly elevated (Cell Phones and Cancer Risk Fact Sheet – NCI).
Million Women Study: No association with glioma, meningioma, or non-CNS tumors; initial acoustic neuroma association was not sustained with longer follow-up (Cell Phones and Cancer Risk Fact Sheet – NCI).
COSMOS Study: Among 264,574 participants, no association with glioma, meningioma, or acoustic neuroma, with median follow-up over 7 years (Cell Phones and Cancer Risk Fact Sheet – NCI).
CERENAT (France): No association for regular users, but heaviest users showed increased glioma and meningioma risks, suggesting a dose-response relationship (Cell Phones and Cancer Risk Fact Sheet – NCI).
Swedish Pooled Analysis: Found increased brain cancer risk for use before age 20, an unexpected detail given children’s theoretical vulnerability due to developing nervous systems (Cell Phones and Cancer Risk Fact Sheet – NCI).
Children’s Studies (CEFALO, MOBI-Kids): No increased cancer risk in children aged 7–19 and 10–24, respectively, despite theoretical concerns (Cell Phones and Cancer Risk Fact Sheet – NCI).
Animal Studies
Animal studies provide some evidence of potential risks, particularly for Schwannomas. The NTP and Ramazzini Institute found small increases in heart Schwannomas in male rats exposed to RF radiation at 3, 6, or 9 watts per kilogram for 18 hours daily, but these findings are limited by species differences and rarity in humans. The NCI notes these results raise questions but do not conclusively link to human brain cancers (Cell Phones and Cancer Risk Fact Sheet – NCI). Another study on 1800 MHz RF-EMF exposure showed inhibited neurite outgrowth in embryonic neural stem cells at 4 W/kg for 3 days, suggesting potential developmental impacts (Exposure to 1800 MHz radiofrequency radiation impairs neurite outgrowth of embryonic neural stem cells | Scientific Reports).
Occupational and Environmental Exposures
For workers with high RF exposure, such as Navy electronics technicians, some studies suggest increased risks of non-lymphocytic leukemia and brain cancer, but results are inconsistent. The NCI’s fact sheet on EMF and cancer notes early studies (1980s–1990s) reported higher cancer rates in electrical occupations, but recent studies with exposure measurements generally show no increased risk (Electromagnetic Fields and Cancer – NCI). Children living near power lines with exposures ≥0.3–0.4 μT showed a 1.4–1.7-fold increased risk of leukemia in pooled analyses, affecting a small percentage (<3%) of children, but mechanisms remain unclear (Electromagnetic Fields and Cancer – NCI).
Expert Opinions and Classifications
The IARC classified RF radiation as “possibly carcinogenic to humans” (Group 2B) in 2011, based on limited evidence from human and animal studies, reflecting ongoing controversy (Cell Phones and Cancer Risk Fact Sheet – NCI). The American Cancer Society, NIEHS, FDA, CDC, FCC, and European Commission (2015) generally conclude no definitive evidence of harm, with calls for more research. The European Commission found no increased brain tumor risk from RF exposure but noted an open question for acoustic neuroma (Electromagnetic Fields and Cancer – NCI).
Frequency Bands and Exposure Levels
Cell towers emit RF in bands like 850 MHz, 1900 MHz, and higher for 5G, with exposure measured by Specific Absorption Rate (SAR), typically below 0.1 W/kg in studies, far below thermal effect thresholds. The FCC provides SAR data for devices, and the NCI notes exposures are generally within safety limits (Cell Phones and Cancer Risk Fact Sheet – NCI).
Avoidance Guidelines
Given the uncertainty, several practical measures can reduce exposure, though their effectiveness in preventing cancer is not proven:
Reduce Call Time: Limit duration of calls, especially long conversations.
Use Speaker Mode or Headphones: Use wired headphones or speaker mode to increase distance from the head.
Avoid Calls in Weak Signal Areas: Weak signals increase emission power, so prefer texting or waiting for stronger signals.
Maintain Distance from Cell Towers: Avoid prolonged proximity to cell towers, though residential exposure is typically low.
The evidence suggests a possible link between long-term EMF/RF exposure and brain cancers/Schwannomas, but it is not conclusive, with significant controversy. Human studies often show no clear association, while animal studies and some occupational data raise concerns. This may be due to differential synergist like viral exposures exposure histories alone [J. Jyrkkanen 2025, comm.]. Avoidance guidelines offer precautionary measures, though their impact on cancer risk reduction is uncertain. Given the 2025 context, ongoing research, such as the COSMOS study, continues to monitor long-term effects, emphasizing the need for balanced public health approaches.
My Lifetime Research Findings:After reading the following and following the links if you want further reading of my literature, try using these search terms after my name: Jorma Jyrkkanen antibiotics, cancer, oncology, cardiology, epigenetics, music, Jormawankenobe, birds, hiking, climate, virology, covid, spike, BWC, geopolitics, WEF, bombing, Deep State, evolution, Darwin, spallation, methylation, bald eagles, Khutzeymateen, Kvenland, extinction, Skagit, Books, blogspot.com, twitter, wordpress.com and follow the links. You may also explore my Bibliography which I add to on occasion. https://jormajyrkkanen.ca/2024/02/14/jorma-antero-jyrkkanens-bibliography-2024-02-14/
3. Ontario Minnesota Pulp and Paper company were liquidating the Ontario boreal forests by fudging regeneration statistics. Chief Forester George Brown shut them down and made them agree to Boreal Forest Clear cut Guidelines and regeneration plans.
4. Vision herbicide, a glyphosate Monsanto product, had 350 ppm of carcinogenic 1,4-dioxane in it and its surfactant polyoxyethyleneamine POEA added to its carcinogenic risk. https://jorma-jyrkkanen.livejournal.com/184872.html
Spallation synthesis of Boron probably made life possible in the universe. It seems we owe our existence to high energy radiation as well as star stuff.
My Conclusion: Cosmic ray spallation is thought to be responsible for the abundance in the universe of some light elements—lithium, beryllium, and boron—as well as the isotope helium-3. They exist only by virtue of their nucleosynthesis during the Big Bang, and also cosmic ray spallation. These lighter elements in the present universe are therefore thought to have been produced through thousands of millions of years of cosmic ray (mostly high-energy proton) mediated breakup of heavier elements, spallation in interstellar gas and dust. This suggests life owes its existence much to spallation by proton radiation collaborating with star stuff.
Pro-vaccination Dr blows whistle on vaccination and finds huge DNA contamination which can increase inflammation and myocarditis, SV40 linked to brain cancer, bone, NHL and malignant mesothelioma and spike protein in heart muscle on autopsy and batch switch from approved to mass produced for public use.
8. I gave evidence at in International Conference on Genetics, Genetic disorders and Stem Cells in Stockholm in 2018 that there is a pandemic of cancers caused by infectious agents ongoing and mouse mammary tumor virus (MMTV), simian virus 40 (SV40) needed to be added to the list of agents and Epstein Barr virus needs special attention. I computed a rough estimate of surplus cancer deaths worldwide attributable to infectious agents without considering gain-of-function bioweapons of approximately 2,365, 920 Deaths in 2023. This is now of course an under-estimate because we need to add the mortality from Turbo cancers from Covid-19 reported by Japanese, UK and American Oncologists. AIDS cancers ex: lung, anal, hepatocellular, Hodgkins lymphoma, oropharyngeal cancers, sars2 covid-19 cancers, SV40 cancers ex: SV40 may contribute to the development of mesotheliomas in humans. Epidemiological studies are needed. SV40, a monkey and now human polyomavirus, was introduced into the human population as a contaminant of vaccines. Merck Virologist Maurice Hilleman bragged about spreading SV40 tumors to Russian Olympians so America would win the Olympics.
I hereby declare that there is a global pandemic of cancers caused by infectious agents, most natural, some with possible increased pathogenicity by gain-of-function and enhanced transmissability (ex: HIV and SV40 by plandemic or injection). There is an increased risk of developing cancer from exposure to these pathogens and also transmit that risk to others by poor hygeine.
Microbiologist Kevin McKernan ran tests on covid vaccine approved and injected and found injected vaccine had DNA contamination and from sequence data was not the one that was approved.
Breaking News Pfizer Lawyer States We did no commit fraud, we delivered the Fraud the Government Ordered. Covid was a DOD fraud all along leading to over 17 Million Killed Worldwide.
Quoting Dr. David Martin: Peter Daszak wrote in 2015 ‘they needed the media to hype up the need for coronavirus vaccines to sell them’. The very next year Dr Ralf Baric wrote in 2016: ‘..’the (WIV) virus was human ready’ a clear indication a weapon was being readies for human transmission. [Seems to me J. Jyrkkanen ‘Collusion and Conspiracy to commit a bioweapon convention violation BWC for profit’]
21. How Prince Phillip, The Duke of Edinborough and I deliver the Khutzeymateen Grizzly Sanctuary to the World. I had to put this in because its such a good news story I was involved in.
For my conservation and habitat work I was given the honor of having a BC Park Named after me. I asked them to use my nic-name at work “FINN”. Ergo ‘FINN CREEK PARK’.
22. I computed the eigenvectors and principle eigenvalue of Squamish Wintering Bald Eagles to what I think is a close approximation from real data I happily gathered over four years and believe have nailed it. It tells me eagles have evolved a density dependent feedback system of population regulation that leads to growth and a stable age distribution. I Post it because its cool and used my UBC Zoology training beautifully. I think I deserve a PHd for this.
Computing Eigenvalues and Eigenvectors for Squamish River Wintering Bald Eagles. Jorma Jyrkkanen, jjyrkkanen76@outlook.com 3 Dec 2018 Background I fiddled with my 4 years of Squamish Wintering Eagles demographic data and fudged a bit of the model to add males in the Reproduction and got an interesting growth rate, lambda1 and a great Eigenvector or stable age distribution. Here it is. Will play with it until I am happy with data fit and may extend it out to 15-20 years age classes. It’s a dynamic model amenable to gaming based on thousands of field observations.
Discussion What I saw from year to year though were what looked like compensatory density dependent adjustments in age specific survival so it flip flopped p due to competition from yr to yr.
This fact is not amenable to deterministic Markov models like Leslie UNLESS you ‘game it’ or program in ‘if Sxi > or < Sxi critical-then-change-Sxi+ or-n’ ie compensatory survival feedbacks to other age classes. I urge population biology students to try it and watch amazing responses to population and age structure. I used an online Calculator bandicoot.maths.adelaide page written by Mathew Roughan.
Figure 1. Sample Leslie Matrix and Start Population Vector with test age specific reproductive rates for a Female Population based on Hypothetical Statistics to Observe Deterministic Model Responses in Total Population, Age Classes and Growth Rates (Primary Eigenvalue) and proportional age class structure (Eigenvectors).December 3, 2018Created by Jorma A. Jyrkkanen Note. The last three age classes are all adults and there is some presumption of productivity from four year class Bald Eagles ie R4=0.1
Eagles Run 2 with a Working Survivorship Population Vector but discrepancy in observed and model that works.
This latter run has a high crash potential (CP) in 8 yrs and a lot of fluctuation. For a more realistic model I have to incorporate my field data.
The bottom box has the stable age structure eigenvector and age specific eigenvalues. The latter more realistic run works but stability hangs on a few percentage survivorships of juveniles age classes and only includes 8 Age classes total.
Conclusion I concluded that the dynamic nature of feedbacks assures high survival of recruits to replace low adult mortality. Note how with even static Survivorships (Sij diagonals in L) there is an innate generation of wild fluctuations in age-class abundance in the curves at the bottom but I caution that in the field this is difficult to tease apart from random effects of migration linked as it is to weather and food availability. This phenomenon no doubt leads a lot of people to think a population is in trouble when only short-term observations are made and it is not true. Time lags are working to smooth out the age class distributions and real population. You only come to know these statistics as probably true after years of observation. I did 4 with thousands of observations and it was just starting to sink in. Keep in mind that these parameters apply only to the female side of the population. If you add the males it will approximately double.
I was a herd animal til I had a good look around. The Bulls liked the Lambs best.
FUNDING: I only received remuneration on discoveries 1 and 3. The remainder of all my scientific work and the thousands of hours I put in to them has been paid for by me and the hardship it imposed on my family. If some philanthropist has funds to chip in and reimburse me for my work for humanity and nature it would be helpful. My situation is very humble and I could do more with help. To help contact me by text message and we can talk on telegram.
I dedicate my life’s research to my Father and Mother who gave me life. Dad owed his life to a Railway spike. Attacked by Russians he ran out of bullets and a Soviet soldier came to shoot him but dad got him in the heart with a long thrown railway spike, took his firearm and continued the battle which the Finns won. None of my work would have happened without his strength and courage and a railway spike.
Immunity and mitochondria are interlinked with each other. An important feature of mitochondria is it can regulate activation, differentiation, and survival of immune cells. In addition, it can also release signals such as mitochondrial DNA (mtDNA) and mitochondrial ROS (mtROS) to regulate transcription of immune cells. Risk factors for mitochondrial dysfunction and by proxy the immune system include antibiotics, pesticides and covid and spike protein. mitochondria have emerged as signaling organelles that contribute critical decisions about cell proliferation, death, and differentiation. Mitochondria not only sustain immune cell phenotypes but also are necessary for establishing immune cell phenotype and their function. Mitochondria can rapidly switch from primarily being catabolic organelles generating ATP to anabolic organelles that generate both ATP and building blocks for macromolecule synthesis. This enables them to fulfill appropriate metabolic demands of different immune cells. Mitochondria have multiple mechanisms that allow them to activate signaling pathways in the cytosol including altering in AMP/ATP ratio, the release of ROS and TCA cycle metabolites, as well as the localization of immune regulatory proteins on the outer mitochondrial membrane. Mitochondria are hubs in cellular signaling and produce ROS that drive production of inflammatory cytokines and play a role in removing bacteria.
Personal Page of Jorma Jyrkkanen, Composer, Author, Researcher 